MORPHOLOGICAL CHANGES IN THE HEART UNDER EXPERIMENTAL HYPOKINESIA
Abstract
This article analyzes the morphological changes in the heart caused by physical inactivity (hypokinesia), their pathophysiological mechanisms, and clinical significance based on international sources. Studies show that hypokinesia promotes myocardial dilatation, hypertrophy, and interstitial fibrosis processes. Consequently, the heart's pumping function declines, increasing the risk of heart failure, arrhythmias, and other cardiovascular diseases. These processes are visually represented through graphical data.
Keywords
Hypokinesia, Morphological changes in the heart, Interstitial fibrosis, Myocardial dilatation, Cardiac hypertrophy, Pathophysiological mechanisms, Metabolic dysfunction, Oxidative stress, Mitochondrial dysfunction, Inflammatory processes, Apoptosis, Heart failure, Arrhythmias, Microscopic changes, Impact of physical activity on the heart, Regular physical activity, Cardiovascular diseases, Clinical outcomes, Experimental research, Preventive measures.How to Cite
References
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